Short-term calorie restriction and 17α-estradiol administration elicit divergent effects on proteostatic processes and protein content in metabolically active tissues.
Nicotinamide mononucleotide (NMN) supplementation rescues cerebromicrovascular endothelial function and neurovascular coupling responses and improves cognitive function in aged mice.
Mitochondrial oxidative stress impairs contractile function but paradoxically increases muscle mass via fibre branching.
Independent effects of dietary fat and sucrose content on chondrocyte metabolism and osteoarthritis pathology in mice.