An altered immune response to Epstein-Barr nuclear antigen 1 in pediatric systemic lupus erythematosus.
Humoral antigenic targets of the ribosomal P0 lupus autoantigen are not limited to the carboxyl region.
Modification of lupus-associated 60-kDa Ro protein with the lipid oxidation product 4-hydroxy-2-nonenal increases antigenicity and facilitates epitope spreading.