Selective and marked blockade of endothelial sprouting behavior using paclitaxel and related pharmacologic agents.
Genomic locus proteomic screening identifies the NF-κB signaling pathway components NFκB1 and IKBKG as transcriptional regulators of Ripk3 in endothelial cells.
An inhibitor of endothelial ETS transcription factors promotes physiologic and therapeutic vessel regression.
The chromatin-remodeling enzyme CHD3 plays a role in embryonic viability but is dispensable for early vascular development.
