Dysregulated long non-coding RNA in Sjögren’s disease impacts both interferon and adaptive immune responses.
Author Correction: Genome-wide association study identifies Sjögren’s risk loci with functional implications in immune and glandular cells.
Genome-wide association study identifies Sjögren’s risk loci with functional implications in immune and glandular cells.
And Yet It Moves: Oxidation of the Nuclear Autoantigen La/SS-B Is the Driving Force for Nucleo-Cytoplasmic Shuttling.
Defective Efferocytosis in a Murine Model of Sjögren’s Syndrome Is Mediated by Dysfunctional Mer Tyrosine Kinase Receptor.