Ablation of intestinal epithelial sialylation predisposes to acute and chronic intestinal inflammation in mice.
Endothelial Slc35a1 deficiency causes loss of LSEC identity and exacerbates neonatal lipid deposition in the liver in mice.
Novel GNE missense variants impair de novo sialylation and cause defective angiogenesis in the developing brain in mice.
Clearance of VWF by hepatic macrophages is critical for the protective effect of ADAMTS13 in sickle cell anemia mice.
