Oklahoma Nathan Shock Aging Center – assessing the basic biology of aging from genetics to protein and function.
Determining the contributions of protein synthesis and breakdown to muscle atrophy requires non-steady-state equations.
Systemic delivery of a mitochondria targeted antioxidant partially preserves limb muscle mass and grip strength in response to androgen deprivation.
A Novel Stable Isotope Approach Demonstrates Surprising Degree of Age-Related Decline in Skeletal Muscle Collagen Proteostasis.
Age-related susceptibility to muscle damage following mechanotherapy in rats recovering from disuse atrophy.
Tumor burden negatively impacts protein turnover as a proteostatic process in non-cancerous liver, heart, and muscle, but not brain.
